Yeah, I can't remember the last time I saw 93% ground beef at 2.99. I just got back from the store, bought 90% ground beef at 6.29 per pound, that is about normal. It occasionally goes on sale but never that low. Chicken is out fucking rageous, especially if you are going for organic/natural/whatever. Shit is like 10 per pound. Apples were 4.99 per pound. On sale. Bananas are cheap as fuck though. This is right outside of DC, which is expensive, but less expensive than NYC I would imagine.Eating healthy is expensive because you're buying a $5 bag of apples for fucking applesauce. How about instead you buy whatever fruit is on sale for $1.29 per pound or cheaper? Also for morning meals Bananas are great, and here in NY it's 77 cents per pound. You can get a week's worth of fruit for $5.00.
It's honestly about smart shopping. Buy family packs of meat, especially when they're on sale. Chicken should be $1.99-2.99 per pound. 93% Beef at $2.99 per pound. I don't wait for sales on ground turkey because their usually isn't any. So I buy the family pack at $7.99.
These are the veggies I buy that last me a week. $6.99
That doesn't even make any sense...
Jesus fuck you're stupid. Butter isn't "good fat." I can't believe the rank idiocy in this thread.I like the big bag of veggies. You just steam them or what?
You can get coconut oil online at iherb.com. Not sure on the butter I just use regular. I never used iherb but I've had people tell me it's a good site. I cant remember what store I got mine at right now, wasnt whole foods or trader joes. Maybe Fairway Market? Anyway I use this:
http://www.iherb.com/Nutiva-Organic-...oz-858-ml/4716
Good fat: avocado, macadamia nuts (buy from costco or similar, they rape you on the price at other stores), olive oil, butter/clarified butter (never tried clarified myself)
-BACKGROUND:
Substitution of dietary polyunsaturated for saturated fat has long been recommended for the primary prevention of cardiovascular disease (CVD), but only a few prospective cohort studies have provided support for this advice.
METHODS:
We assessed the association of dietary linoleic and total polyunsaturated fatty acid (PUFA) intake with cardiovascular and overall mortality in a population-based cohort of 1551 middle-aged men. Dietary fat composition was estimated with a 4-day food record and serum fatty acid composition.
RESULTS:
During the 15-year follow-up, 78 men died of CVD and 225 of any cause. Total fat intake was not related to CVD or overall mortality. Men with an energy-adjusted dietary intake of linoleic acid (relative risk [RR] 0.39; 95% confidence interval [CI], 0.21-0.71) and PUFA (RR, 0.38; 95% CI, 0.20-0.70) in the upper third were less likely to die of CVD than men with intake in the lower third after adjustment for age. Multivariate adjustment weakened the association somewhat. Mortality from CVD was also lower for men with proportions of serum esterified linoleic acid (RR, 0.42; 95% CI, 0.21-0.80) and PUFA (RR, 0.25; 95% CI, 0.12-0.50) in the upper vs lower third, with some attenuation in multivariate analyses. Serum and to a lesser extent dietary linoleic acid and PUFA were also inversely associated with overall mortality.
CONCLUSIONS:
Dietary polyunsaturated and more specifically linoleic fatty acid intake may have a substantial cardioprotective benefit that is also reflected in overall mortality. Dietary fat quality seems more important than fat quantity in the reduction of cardiovascular mortality in men.
-OBJECTIVE:
To investigate whether quantity or quality of dietary fat predicts coronary heart disease (CHD) events in middle-aged type 2 diabetic subjects.
RESEARCH DESIGN AND METHODS:
The dietary habits of 366 type 2 diabetic men and 295 women, aged 45-64 years and free from CHD, were assessed with a 53-item food frequency questionnaire. They were followed up for 7 years.
RESULTS:
Men in the highest tertile of the polyunsaturated/saturated fat (P/S) ratio (>0.28) had a significantly lower risk for CHD death than men in the two lowest tertiles (5.0 vs. 14.2%, P = 0.009). The risk for all CHD events was 14.2 vs. 23.2%, respectively (P = 0.044). P/S ratio did not predict CHD events in women. In Cox multiple regression analyses taking into account other cardiovascular risk factors, the highest P/S ratio tertile was associated with the lowest rate of CHD death in men (P = 0.048).
CONCLUSIONS:
Low P/S ratio in men predicted future CHD events in type 2 diabetic subjects independently of conventional CHD risk factors.
-Our findings suggest that replacing saturated and trans unsaturated fats with unhydrogenated monounsaturated and polyunsaturated fats is more effective in preventing coronary heart disease in women than reducing overall fat intake.
-The multivariate RR for a 1% energy increase from stearic acid was 1.19 (95% CI: 1.02, 1.37). The ratio of polyunsaturated to saturated fat was strongly and inversely associated with CHD risk (multivariate RR for a comparison of the highest with the lowest deciles: 0.58; 95% CI: 0.41, 0.83; P for trend < 0.0001). Conversely, higher ratios of red meat to poultry and fish consumption and of high-fat to low-fat dairy consumption were associated with significantly greater risk.
Background: Saturated fatty acid (SFA) intake increases plasma LDL-cholesterol concentrations; therefore, intake should be reduced to prevent coronary heart disease (CHD). Lower habitual intakes of SFAs, however, require substitution of other macronutrients to maintain energy balance.
Objective: We investigated associations between energy intake from monounsaturated fatty acids (MUFAs), polyunsaturated fatty acids (PUFAs), and carbohydrates and risk of CHD while assessing the potential effect-modifying role of sex and age. Using substitution models, our aim was to clarify whether energy from unsaturated fatty acids or carbohydrates should replace energy from SFAs to prevent CHD.
Design: This was a follow-up study in which data from 11 American and European cohort studies were pooled. The outcome measure was incident CHD.
Results: During 4-10 y of follow-up, 5249 coronary events and 2155 coronary deaths occurred among 344,696 persons.For a 5% lower energy intake from SFAs and a concomitant higher energy intake from PUFAs, there was a significant inverse association between PUFAs and risk of coronary events (hazard ratio: 0.87; 95% CI: 0.77, 0.97); the hazard ratio for coronary deaths was 0.74 (95% CI: 0.61, 0.89).For a 5% lower energy intake from SFAs and a concomitant higher energy intake from carbohydrates, there was a modest significant direct association between carbohydrates and coronary events (hazard ratio: 1.07; 95% CI: 1.01, 1.14); the hazard ratio for coronary deaths was 0.96 (95% CI: 0.82, 1.13). MUFA intake was not associated with CHD. No effect modification by sex or age was found.
Conclusion: The associations suggest that replacing SFAs with PUFAs rather than MUFAs or carbohydrates prevents CHD over a wide range of intakes.
Most of these were prospective cohort studies, you retard. You can't even properly classify them, lol. Sure, epidemiological is the broad category (we say observational...), but come on. If you're going to criticize, t lest be somewhat accurate, dumbshit!still citing epidemiological studies heh
I'm still waiting on your experiment design. You simply "write off" data, and yet accept a ridiculous hypothesis that is not supported by either the data or any professional medical or health organization in the USA. Please provide high quality data supporting this retarded Paleo diet. I'm waiting "lipid researcher" (lol, give me a break!)that's precisely what a cohort study is. these studies have countless undefined variables. what are you having trouble understanding?
signed,
a biochemist/lipid researcher
"Normalizing your system" - spoke like a true "biochemist/lipid researcher"well, if you have fatigue as a result of a metabolic disorder or if you feel like shit due to gluten intolerance, normalizing your system would feel like quite a boost
Oh, I'm definitely no expert, not even close, but compared to scientifically illiterate retards like you and Dashel I'm motherfucking Einstein. You clearly are no "lipid researcher", you brain dead twit.what do i know? you spent years studying outdated information only to get a job prescribing statins (assuming you really are a doctor). seriously, you're not an expert so stop acting like one.
it's not hard to analyze the nutrient profiles of foods and to think critically of the things you're stuffing down your throat. but go ahead and eat your 13 servings of grains.